What Causes Abnormal Vitamin B6 Levels?
To successfully treat and prevent recurrence of abnormal vitamin B6 levels we need to understand and — if possible — remove the underlying causes and risk factors.
We need to ask: "What else is going on inside the body that might allow abnormal vitamin B6 levels to develop?"
Diagnose your symptoms now!
- let The Analyst™ find what's wrong
- learn what you should be doing right now
- identify any nutritional deficiencies
Accurate diagnosis of the factors behind abnormal vitamin B6 levels consists of three steps:
Step 1: List the Possible Causative Factors
Identify all disease conditions, lifestyle choices and environmental risk factors that can lead to abnormal vitamin B6 levels. Here are two possibilities:
- Nutritional Deficiency Anemia
Step 2: Build a Symptom Checklist
Identify all possible symptoms and risk factors of each possible cause, and check the ones that apply:
lighter/paler skin color
moderate alcohol consumption
no desire to eat breakfast
minor joint pain/swelling/stiffness
a few white spots on fingernails
being prone to 'stitches'
high sensitivity to bright light
being a recovered alcoholic
frequent unexplained nausea
severe fatigue after slight exertion
... and so on
Step 3: Rule Out or Confirm each Possible Cause
A differential diagnosis of your symptoms and risk factors finds the likely cause of abnormal vitamin B6 levels:
|Nutritional Deficiency Anemia
* This is a simple example to illustrate the process
Arriving at a Correct Diagnosis
is our online diagnosis tool that learns all about you through a straightforward process of multi-level questioning, providing diagnosis at the end.
Vitamin B6 (pyridoxine). Unit: ng/mL [nmol/L]
→ Don't know
→ Confirmed deficiency in the past
→ Under 4  (low)
→ 4 to 18 [16-73] (normal)
→ Over 18  (elevated)
Based on your response to this question, which may indicate history of B6 deficiency, low B6 levels, normal B6 levels or high B6 levels, The Analyst™
will consider possibilities such as:
A functional pyridoxine deficiency is common in pyroluria (often seen in alcoholics), due not so much to inadequate intake as impaired conversion to its active form, pyridoxal-5-phosphate, and enhanced degradation.
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