The fifth prostaglandin subtype discovered, and usually separated into PGE1 and PGE2. These two, if made by the kidneys, slow sodium reabsorption; if within the uterus, induce a stronger response from less stimulus; if made in the stomach lining inhibit gastric secretion; if secreted by macrophages, target tissues become more accessible to infiltration and inflammation. These are the two prostaglandins whose levels are meant to be stabilized by gamma-linolenic acid (GLA) supplements. Also: PGE

	Premenstrual Syndrome PMS C (Craving) The Condition "...A deficiency of the prostaglandin PGE, in the pancreas and CNS may also be involved in PMS-C..."

Last updated: May 10, 2007